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Topical cyclosporin A as a steroid‐sparing agent for ocular rosacea

baseline. Cox models included age, gender, participating in the population survey, smoking habits, diabetes mellitus, systemic hypertension, ischaemic heart disease and OAG. The ultimate model is presented in Table 1. Higher age and ischaemic heart disease were the only predictors found to be associated with AD. However, there was evidence of interaction between age and gender. Spanning 30 years, this study on a defined population is the longest follow-up study on OAG and the risk of developing AD yet to be reported. Contrary to a recent publication (Lin et al. 2014), OAG was found not to be a risk factor. Two main hypotheses have been proposed to explain the connection between OAG and AD. The first hypothesis includes a common neurodegenerative process involving activation of enzymatic caspases and the production of neurotoxic amyloid b (McKinnon 2003). The second hypothesis implicates optic nerve damage as a consequence of an abnormally highpressure difference across the lamina cribrosa in AD (Wostyn et al. 2009). It is beyond the scope of this report to discuss which of these hypotheses, if any, is most plausible. However, our findings do not corroborate an association between the two disorders.