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Role of sensory neuropeptides in post‐allergic propranolol‐induced bronchoconstriction in guinea pigs in vivo

Background Administration of propranolol can provoke bronchoconstriction in asthmatic patients. We hypothesized that such bronchoconstriction may result from the inflammatory mediators released by an allergic reaction. We recently developed a guinea pig model for propranolol‐induced bronchoconstriction (PIB). Neuropeptides which are released from C‐fibre nerve endings have been postulated to induce bronchial hyperresponsiveness through neurogenic inflammation.