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The role of calcium in the regulation of sugar transport in the pigeon red blood cell.

The saturable, ‘carrier‐mediated’ pathway for sugar transport in pigeon red blood cells may be stimulated by metabolic depletion or by loading the cells with Ca by means of a high‐voltage discharge or the ionophore A 23187. All of these methods for stimulating the saturable pathway of sugar transport also cause a drop in cellular ATP levels. The relationship between the stimulation of transport and the fall in ATP is very similar in metabolically depleted or Ca‐loaded cells, but cells treated with Ca and A23187 show a greater stimulation of transport than would be expected from the decline in ATP. Altering free Ca2+ levels during metabolic depletion has little or no effect on stimulation of the saturable pathway. Conversely, metabolic depletion of fresh cells in Ca‐free solutions has no detectable effect on intracellular free Ca2+ levels. These results suggest that Ca2+ ions are not involved in regulation of this pathway. The non‐saturable pathway for sugar transport is stimulated by a rise in cell Ca. This process is probably stimulated half‐maximally by about 10 microM‐free Ca2+.