{'fraction_non_alphanumeric': 0.031189083820662766, 'fraction_numerical': 0.015594541910331383, 'mean_word_length': 4.648351648351649, 'pattern_counts': {'":': 0, '<': 0, '<?xml version=': 0, '>': 0, 'https://': 0, 'lorem ipsum': 0, 'www.': 0, 'xml': 0}, 'pii_count': 0, 'substrings_counts': 0, 'word_list_counts': {'cursed_substrings.json': 0, 'profanity_word_list.json': 0, 'sexual_word_list.json': 0, 'zh_pornsignals.json': 0}}
Formation of activated oxygen in the hypoxic rat liver

The biliary GSSG efflux rate of normoxic perfused rat liver was 1·5 ± 0·2 nmol/min/g liver wet weight. The GSSG efflux rate as indicator for the flux through the glutathine peroxidase and, therefore, for an oxidative loading incvreased with the extent of hypoxia. 2·6 ± 0·5 nmol/min/g were released from the severely hypoxic liver. The hydroxyl radical scavenger formate as well as the xanthine oxidase inhibitor allopurinol reduced the efflux rate of GSSG.