{'fraction_non_alphanumeric': 0.03676470588235294, 'fraction_numerical': 0.016544117647058824, 'mean_word_length': 5.722222222222222, 'pattern_counts': {'":': 0, '<': 1, '<?xml version=': 0, '>': 0, 'https://': 0, 'lorem ipsum': 0, 'www.': 0, 'xml': 0}, 'pii_count': 0, 'substrings_counts': 0, 'word_list_counts': {'cursed_substrings.json': 0, 'profanity_word_list.json': 0, 'sexual_word_list.json': 0, 'zh_pornsignals.json': 0}}
Determination of the T‐Cell Subset Producing γ‐Interferon in Tuberculous Pleural Effusion

The percentage of cells of different T‐cell subsets and their functions in tuberculous pleural effusion were investigated. The percentage of OKT4‐positive cells was 65 ± 2% (mean ± SEM, n=8) and that of OKT8‐positive cells was 19 ± 3% (n=8). Pleural T lymphocytes of patients with tuberculous pleurisy responded well to stimulation with purified protein derivative of tuberculin, and deoxyribonucleic acid synthesis was observed along with gamma interferon (IFN‐γ) production. When pleural T lymphocytes of patients with tuberculous pleurisy were treated with OKT4 monoclonal antibody and complement, a significant decrease in IFN‐γ production was observed in all eight patients (P<0.005), whereas no definite decrease in IFN‐γ production was found after treatment with OKT8 monoclonal antibody and complement. These results suggest that at least the OKT4+/OKT8− T‐cell subset is responsible for the antigen‐specific IFN‐γ production in pleural T lymphocytes of patients with tuberculous pleurisy.