{'fraction_non_alphanumeric': 0.03898305084745763, 'fraction_numerical': 0.023728813559322035, 'mean_word_length': 5.15625, 'pattern_counts': {'":': 0, '<': 0, '<?xml version=': 0, '>': 0, 'https://': 0, 'lorem ipsum': 0, 'www.': 0, 'xml': 0}, 'pii_count': 0, 'substrings_counts': 0, 'word_list_counts': {'cursed_substrings.json': 0, 'profanity_word_list.json': 0, 'sexual_word_list.json': 0, 'zh_pornsignals.json': 0}}
Effect of single gallium arsenide exposure on some biochemical variables in porphyrin metabolism in rats

Gallium arsenide (GaAs) inhibited, dose dependently, δ‐aminolevulinic acid dehydratase (ALAD) activity in blood (as observed on days 1, 7 and 15), liver and brain (on day 15) after a single oral treatment. Levels of blood zinc protoporphyrin (ZPP) and urinary Δ‐aminolevulinic acid (ALA) excretion were elevated on day 7 and day 15 following exposure to 2000 mg kg−1 GaAs. A dose‐dependent increase in blood As contents was observed while blood Ga was detectable only at higher doses.