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P01.33: The use of the minimum projection mode in four‐dimensional examination of the fetal heart with spatiotemporal image correlation (STIC)

Results: DC was detected in 19 cases (0.3%) and premature closure of FO in 2 (0.03%). Two fetuses with DC presented with ascites and polyhydramnion. In two cases DC coexisted with CALM. Once there was DC of one of the twins. DC was present at mean 33 wks of gestation. In 12 cases there was maternal pharmacotherapy. Abnormal DC was present at the mean on 3rd day of pharmacotherapy. In addition to DC there were also other abnormalities: myocardial hypertrophy, tricuspid valve regurgitation, pulmonary regurgitation, pericardial effusion, or disproportion. Two fetuses presented arrhythmias. Mean 25 days after DC there was or vaginal delivery (10×) or CS (9×). Mean birth weight was 3074, mediana of Apgar score-9. There was 17 survivors. In neonatal echo in all cases still right ventricular hypertrophy was present with no clinical adverse outcome. During the same period 2 cases of premature closure of FO were observed. The first one presented with ascites at 28 wks of gestation and died in utero. The second one presented with acute cardiomegaly, disproportion, right ventricular wall hypokinesis and tricuspid valve regurgitation, had emergency CS at 35 wks of gestation. This baby was discharged home at 15th day of life with no clinical symptoms. Conclusions: 1. Premature DC usually caused by maternal pharmacotherapy with transient myocardial hypertrophy, TR, disproportion, but without cardiomegaly and this anomaly was reversible during fetal life. 2. Premature closure of foramen ovale was very rare phenomenon presented with acute cardiomegaly, right ventricle dyskinesis, TR and should prompt to emergency CS.