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Effects of transitory maternal pteroylglutamic acid (PGA) deficiency on respiratory control ratios, phosphorylative capacity, and cytochrome oxidase activities of mitochondria from rat embryos.

Cytochrome oxidase activities and protein contents of homogenates and mitochondrial fractions of embryos from untreated and 9-methyl PGA treated pregnant rats were determined on various days of gestation. Rates of respiration, ADP:O, and respiratory control ratios were determined with the mitochondrial fractions. The cytochrome oxidase specific activities of homogenates or mitochondrial fractions of treated embryos were similar to those of controls of the same age. However, total mitochondrial protein and cytochrome oxidase activities of the treated embryos were similar to those of controls even though the treated embryos were smaller than controls. The amount of oxygen consumed by the treated embryos was not significantly different from that consumed by controls and the ADP:O and respiratory control ratios calculated for mitochondrial fractions of treated and control embryos were not significantly different.