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Activation of p53 signalling in acetylsalicylic acid‐induced apoptosis in OC2 human oral cancer cells

Background  Nonsteroidal anti‐inflammatory drugs (NSAIDs) such as acetylsalicylic acid (ASA, aspirin) are well known chemotherapeutic agents of cancers; however, the signalling molecules involved remain unclear. The aim of this study was to investigate the possible existence of a putative p53‐dependent pathway underlying the ASA‐induced apoptosis in OC2 cells, a human oral cancer cell line.