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Cardiac troponins T and I before and after renal transplantation.

Cardiac troponins T (cTnT) and I (cTnI), structural proteins of the thin filament, are sensitive markers for myocardial injury (1). In unstable angina, abnormal cTnT and cTnI concentrations have been linked to a high short-term incidence (within 6 months) of adverse cardiac events \[acute myocardial infarction (AMI) and death\] (2). Studies of serum of patients with chronic renal failure have shown that cTnT [Cardiac T; Boehringer Mannheim (BMC), Indianapolis, IN] is abnormally increased more frequently than cTnI, suggesting that cTnT may be more nonspecific (3)(4). High troponin T concentrations in these patient groups could be caused by reexpression of troponin T in regenerating skeletal muscles (as observed by Bodor et al. in patients with Duchenne muscular dystrophy (5)), nonspecificity of the existing cTnT assay, or minor myocardial …