{'fraction_non_alphanumeric': 0.016666666666666666, 'fraction_numerical': 0.002564102564102564, 'mean_word_length': 5.007692307692308, 'pattern_counts': {'":': 0, '<': 0, '<?xml version=': 0, '>': 0, 'https://': 0, 'lorem ipsum': 0, 'www.': 0, 'xml': 0}, 'pii_count': 0, 'substrings_counts': 0, 'word_list_counts': {'cursed_substrings.json': 0, 'profanity_word_list.json': 0, 'sexual_word_list.json': 0, 'zh_pornsignals.json': 0}}
The effect of oral trypsin inhibitor on pancreatic damage in the rat.

Abstract 
A study has been made of the influence of experimentally-induced pancreatic enlargement on pancreatic damage. Soybean trypsin inhibitor was fed to rats in doses sufficient to produce pancreatic enlargement. This agent was given both before and after the production of pancreatic necrosis by oral d,1-ethionine and before the induction of pancreatitis by ligation of the pancreatico-biliary duct. Generally there was no difference between the severity of pancreatic damage in rats fed trypsin inhibitor and in those on control diets. An exception was in rats given d,1-ethionine after treatment with soybean trypsin inhibitor. In these rats histology showed a less severe form of pancreatic necrosis.