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Killing of virus-induced thymic lymphoma cells obtained from primary and early transfers by activated macrophages.

Primary radiation leukemia virus-induced thymic lymphomas were susceptible to both stasis and lysis mediated by LPS-activated macrophages. During short in vitro cultivation the X-irradiation-induced lymphoma (PIR-2) cells were killed to the same extent (up to 99%) by LPS-activated macrophages as the RadLV lymphoma cells at effector:target ratios of 10:1 and 1:1. Since RadLV is not present in PIR-2 lymphoma cells, its presence is not required for susceptibility of the lymphoma cells to macrophage-mediated killing. Our data also disprove the assumption that artifacts of long-term cultivation in vitro are responsible for tumor susceptibility to cytostasis and cytolysis mediated by activated macrophages.