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Abnormal connexin expression in human chronic wounds

Regulated alteration of connexin expression has been shown to be integral to acute wound repair. Downregulation of the gap‐junction protein connexin 43 at the wound edge has been correlated with keratinocyte and fibroblast migration, while abnormal overexpression of connexin 43 significantly perturbs healing, as shown in the streptozotocin diabetic rodent impaired healing model.