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Modeling prosopagnosia: computational theory and experimental investigations of a deficit in face recognition

Prosopagnosia is defined as a profound deficit in facial identification which can be either acquired due to brain damage or is present from birth, i.e. congenital. Normally, faces and objects are processed in different parts of the inferotemporal cortex by distinct cortical systems for face vs. object recognition, an association of function and location. Accordingly, in acquired prosopagnosia locally restricted damage can lead to specific deficits in face recognition. However, in congenital prosopagnosia faces and objects are also processed in spatially separated areas. Accordingly, the face recognition deficit in congenital prosopagnosia can not be solely explained by the association of function and location. Rather, this observation raises the question why and how such an association evolves at all. So far, no quantitative or computational model of congenital prosopagnosia has been proposed and models of acquired prosopagnosia have focused on changes in the information processing taking place after inflicting some kind of “damage” to the system. To model congenital prosopagnosia, it is thus necessary to understand how face processing in congenital prosopagnosia differs from normal face processing, how differences in neuroanatomical development can give rise to differences in processing and last but not least why facial identification requires a specialized cortical processing system in the first place.