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Embryonic miRs to the Rescue.

The advent of reperfusion therapies has led to better outcomes and improved survival after myocardial infarction (MI), but the pathophysiological effects of MI can be long-lasting. Widespread cardiomyocyte apoptosis and necrosis, coupled with a limited reparative capacity of injured cardiac tissue, leads to adverse ventricular remodeling and heart failure (HF). Currently, treatment strategies for HF are limited and do not correct the underlying damage with transplant being the only curative option. Thus, HF following MI is a leading cause of morbidity and mortality worldwide.