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The involvement of plasma free fatty acids in (+)‐amphetamine‐induced hyperthermia in rats

a highly significant increase of 38.8% in brain noradrenaline over those receiving only the amine (Table 1). Experiments still in progress indicate that DMSO plus dopamine intensify fixed staring and catatonia that occur after administration of this amine alone, although the associated amine brain levels have not yet been determined. Results indicate that DMSO, a solvent having both polar and nonpolar characteristics, facilitates entry of adrenaline, noradrenaline and possibly dopamine across the blood-brain barrier. The occurrence of this phenomenon may, in part, be related to the ability of this solvent, acting as a carrier of biogenic amines, to traverse both aqueous and lipid phases or components of the barrier. These findings may have significance in conditions requiring brain amine replenishment.