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NaHS ameliorates diabetic vascular injury by correcting depressed connexin 43 and 40 in the vasculature in streptozotocin‐injected rats

Objectives Cardiovascular complication contributes an important role to morbidity and mortality in patients with diabetes. We hypothesized that these abnormalities are mainly mediated by oxidative stress, endothelial dysfunction and impaired intracellular communications. Thus, we examined vasoactivity and expression of connexin (Cx) 43 and 40, protein kinase C‐ɛ (PKCɛ) and NADPH oxidase of the vasculature of thoracic aorta in streptozotocin (STZ)‐injected rats, and whether NaHS could reverse these abnormalities compared with aminoguanidine.