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Failure to demonstrate monoamine oxidase inhibition by glyceryl trinitrate in vivo.

Glyceryl trinitrate (GTN) or the monoamine oxidase (MAO) inhibitor nialamide was given to rats or mice which then received 14C-tyramine. Nialamide but not GTN increased the yield of the 14C-octopamine formed in the heart. In nialamide pretreated mice GTN possibly caused a slight increase in 14C-octopa-mine. GTN given before or after 3H-noradrenaline had no effect on the uptake and retention of this amine in the mouse heart. Neither did it affect the uptake of 3H-metaraminol in this organ. It is concluded that GTN has no effect on the MAO responsible for the in vivo metabolism of tyramine and noradrenaline. The existence of MAO systems with different substrate and inhibitor sensitivity is discussed.