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OCT1 genetic variants are associated with long term outcomes in imatinib treated chronic myeloid leukemia patients

One third of CML patients treated with first line imatinib have suboptimal responses or treatment failures with increased risk for disease progression. Imatinib is actively transported into cells by the SLC22A1 transporter (hOCT1) and its genetic variants may affect intracellular drug import. We studied the effect of SLC22A1 genetic variants on long‐term outcomes of imatinib treated patients.