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Glutamate receptors in the postmortem striatum of schizophrenic, suicide, and control brains

Previous postmortem studies of glutamate receptors and uptake sites have shown decreased D‐aspartate (D‐Asp) (a marker for the high affinity glutamate uptake site) and elevated (+)‐5‐methyl‐10,11‐dihydro‐5H‐dibenzo [a,d]cyclohepten‐5,10‐imine maleate (MK‐801) binding in the putamen in schizophrenia and elevated α‐amino‐3‐hydroxy‐5‐methyl‐4‐isoxazolepropionic acid (AMPA) receptor binding in the caudate nucleus of schizophrenics who committed suicide. The relative effects of schizophrenia, suicide, and neuroleptic treatment in these findings is unclear. This study further explores binding to glutamate receptors (NMDA, kainic acid, and AMPA) and uptake sites in postmortem striatal structures in schizophrenics relative to three control groups (normal controls, neuroleptic‐treated controls, and nonpsychotic suicides).