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Adenosine formation and metabolism during adenosine triphosphate catabolism in Ehrlich ascites tumor cells.

Adenosine formation and metabolism were measured during the course of 2-deoxyglucose-induced adenosine triphosphate (ATP) catabolism. Adenosine did not accumulate unless coformycin, an inhibitor of adenosine deaminase, was present. Following formation of adenylate from ATP, there are two routes by which adenylate can be converted to inosine: by dephosphorylation to adenosine followed by deamination and by deamination to inosinate followed by dephosphorylation. The major route of catabolism of adenylate formed from ATP was deamination, but up to 18% of the adenylate was catabolized by dephosphorylation; the concentration of ATP had no detectable influence on the relative rates of the alternative pathways. Adenosine formed by dephosphorylation of adenylate may also be rephosphorylated and the rate of rephosphorylation is governed by the intracellular concentration of ATP.