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Prognostic factors for survival in cervical cancer--warts and all.

Molecular and epidemiologic studies provide strong evidence of the causative role of specific human papillomaviruses (HPVs) in the development of cervical cancers (/). Laboratory studies have identified the viral E6 and E7 oncogenes as critically important in cell transformation and immortalization, and the E6 protein binding of P53 and E7 complexing with retinoblastoma proteins are central to current theories of HPV-related carcinogenesis (2,3). Specific antiviral therapies and preventive strategies are exciting prospects in the control of cervical cancer, which is globally the second most common cancer in women.