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Correction: Effect of terbutaline sulphate in chronic “allergic” cough

potassium.2 Furthermore, restoration of serum potassium concentration cannot return intracellular values to normal unless mechanisms maintaining the gradient of potassium across the cell membrane are functioning normally; magnesium depletion occurs in patients taking thiazides 222' and impairs the activity of sodium-potassium adenosine triphosphatase. It is important to note, in any case, that the clinical significance of thiazide induced ventricular extrasystoles is not clear, although the association noted between thiazide administration and special forms of extrasystoles such as multifocal beats, couplets, R on T beats, and bigeminy may be important. Insurance statistics and epidemiological data suggest that otherwise healthy people with ventricular extrasystoles found in resting electrocardiograms have a definitely increased risk of sudden death,24 2 and ventricular extrasystoles found during ambulatory monitoring appear to identify a group with an increased prevalence of coronary heart disease and risk of sudden deatb.26 It seems certain that if the metabolic effects of thiazide diuretics do in fact increase the risks of coronary heart disease mortality or of sudden death the increase would be detectable only in a large placebo controlled trial. The only relevant study whose results are still awaited is the Medical Research Council's mild hypertension trial; possibly even the results of this large and long term study might not be definitive on this point, but none the less these data are likely to be the most useful available for estimating the importance of findings of the kind given in the present paper.