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Effects of a Monoclonal Antibody Raised Against Nerve Growth Factor on Skeletal Pain and Bone Healing After Fracture of the C57BL/6J Mouse Femur

A closed femur fracture pain model was developed in the C57BL/6J mouse. One day after fracture, a monoclonal antibody raised against nerve growth factor (anti‐NGF) was delivered intraperitoneally and resulted in a reduction in fracture pain‐related behaviors of ∼50%. Anti‐NGF therapy did not interfere with bone healing as assessed by mechanical testing and histomorphometric analysis.