{'fraction_non_alphanumeric': 0.026836158192090395, 'fraction_numerical': 0.0014124293785310734, 'mean_word_length': 5.445454545454545, 'pattern_counts': {'":': 0, '<': 0, '<?xml version=': 0, '>': 0, 'https://': 0, 'lorem ipsum': 0, 'www.': 0, 'xml': 0}, 'pii_count': 0, 'substrings_counts': 0, 'word_list_counts': {'cursed_substrings.json': 0, 'profanity_word_list.json': 0, 'sexual_word_list.json': 0, 'zh_pornsignals.json': 0}}
Determination and Analysis of the Dynamic Mechanisms That Precede Long-Term Depression in Cerebellar Purkinje Cells

One cellular basis for learning is the phenomenon of synaptic plasticity that has been observed experimentally in neurons. An important form of synaptic plasticity related to motor-learning tasks such as the vestibular-ocular reflex, eye-blink conditioning, and motor coordination is observed in cerebellar Purkinje cells [1]. This particular form of synaptic plasticity, known as long-term depression (LTD), is a lasting decrease in the activity of the synapses between spines on the Purkinje cell dendrites and axons of neighboring granule cells, often referred to as parallel fibers (PF).