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New Perspectives in the Treatment of Neurocritical Patient

2. Our knowledge of pathophysiology has improved beyond primary damage. We know better the mechanisms that are triggered in each acute neurological injury causing secondary damage. From the different inflammatory mechanisms; alterations in Cerebral Blood Flow (FSC) due to loss of selfregulation, and its particular temporal behavior in each particular pathology [1,2]; to the intricate mechanisms of apoptosis. Thus, we have also generated new knowledge about the interrelationships that produce systemic impact in the face of the different Neurological insults [3].