1.0alpha7.dev.108.sent
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Thus, is it that the Igs bind to an autoantigen(s) on specific neurons that ‘subsequently’ show signs of degeneration, to eventually express caspase 3 and die, due to the Ig binding (the AD autoimmune hypothesis), or  alternatively, do the dying neurons express a ‘stress’ protein(s) that the Igs inconsequentially bind (alternative Ig explanation)?