This aggregation of triglycerides occurs through the increase in the synthesis of triglyceride production. Metformin modestly reduces LDL and triglyceride levels. Efficient absorption of dietary fats is dependent on the action of pancreatic triglyceride lipase. Emerging evidence associates FXR activation with alterations in triglyceride metabolism, glucose metabolism, and liver growth. Gastric lipase is not the primary lipase needed for the majority of triglyceride hydrolysis. In triglyceride form, lipids cannot be absorbed by the duodenum. Linseed oil is a triglyceride, like other fats. Statins are not better than fibrates when lowering triglyceride levels. Apo-CIII delays the catabolism of triglyceride rich particles. FXR has also been found to be important in regulation of hepatic triglyceride levels. The word "triacylglycerol" is sometimes used synonymously with "triglyceride". The triglyceride derived from three molecules of stearic acid is called stearin. The phosphatidic acid is also a precursor for triglyceride biosynthesis. K cells secrete gastric inhibitory peptide, an incretin, which also promotes triglyceride storage. It may also increase triglyceride levels.