Macrophages remove senescent erythrocytes, leukocytes, and megakaryocytes by phagocytosis and digestion. Effect of corticosteriod therapy on the phagocytosis of antibody-coated platelets by human leukocytes. It is expressed by many cell types, including epithelial cells, endothelial cells and leukocytes. It is also characterized by invasion of the thyroid tissue by leukocytes, mainly T-lymphocytes. When activated, leukocytes bind to endothelial cells via ICAM-1/LFA-1 and then transmigrate into tissues. Here all erythrocytes, platelets, and most leukocytes form in adults. APC has anti-inflammatory effects on endothelial cells and leukocytes. The principle of their operation is based on the detection of granulocytes esterases, including leukocytes. The deficiency causes ineffective migration and phagocytosis for impacted leukocytes. Teicoplanin is much better at penetrating into leukocytes and phagocytes than vancomycin. It is most commonly caused by antibodies directed against donor leukocytes and HLA antigens. Selectins are adhesion molecules that facilitate the capture of circulating leukocytes. SK1 is highly expressed in spleen, lung and leukocytes. Interactions of actin, myosin and an actin-binding protein of chronic myelogenous leukemia leukocytes. These mediators contribute to inflammation by attracting other leukocytes.