Damage causing agnosia usually occurs in either the occipital or parietal lobes of the brain. Some neurons in the posterior parietal cortex are modulated by intention. Working memory manipulation involves the DLPFC, inferior frontal gyrus, and areas of the parietal cortex. The VA projects widely across the inferior parietal and premotor cortex. It affects the subcortical white matter, particularly that of the parietal and occipital lobes. This is usually due to lesions in the posterior parietal lobe. Portions of the parietal lobe are involved with visuospatial processing. Neglect is most closely related to damage to the temporo-parietal junction and posterior parietal cortex. Thus frontal and parietal bones are purely membranous. Several areas of the parietal lobe are important in language processing. Other theories suggest that distorted body image perceptions stem from within the parietal lobe. It is caused by damage to the parietal lobe of the brain. Histamine also stimulates H+ secretion from parietal cells. The ACA transmits blood to the frontal parietal. It is thinner than the parietal peritoneum.