The damage to the cerebellum or the inferior cerebellar peduncle can cause ataxia. Cerebellar or basal ganglia etiology is also possible. The only excitatory neurons present in the cerebellar cortex are granule cells. The pons is connected to the cerebellum by the cerebellar peduncles. Ataxic cerebral palsy is caused by damage to cerebellar structures. Mutations in NF2 are commonly expressed in meningiomas located in the cerebral and cerebellar hemispheres. They terminate in the hind limb region of the cerebellar cortex. The cerebellar cortex is divided into three layers. The top, outermost layer of the cerebellar cortex is the molecular layer. The pons co-ordinates activities of the cerebellar hemispheres. The fibers arise from the deep cerebellar nuclei. It is caused by lesions of the cerebellar hemispheres. Gaze-evoked nystagmus and other ocular signs of cerebellar dysfunction are common. Errors in reaching are commonly found in patients with cerebellar degeneration. There is no standard course of treatment for cerebellar hypoplasia.