Treatment for lead poisoning normally involves the administration of dimercaprol and succimer. It is effectively treated with British anti-lewisite (dimercaprol). Calcium EDTA is also effective if administered four hours after the administration of dimercaprol. Gold toxicity can be ameliorated with chelation therapy with an agent such as dimercaprol. Her sweat glands excrete dimercaprol and chloramine to counteract blistering agents such as mustard gas. Medications used include dimercaprol, edetate calcium disodium, and succimer. They are related to dimercaprol, which is another chelating agent. This complex is inhibited by dimercaprol (British Antilewisite, BAL), Napthoquinone and Antimycin. For lead encephalopathy it is typically used together with dimercaprol. Poisoning may be treated with dimercaprol; however, evidence for benefit is unclear. Dimercaprol used alone may also redistribute lead to the brain and testes. Dimercaprol is considerably more toxic than succimer. Dimercaprol was first made during World War II. Dimercaprol works by binding with heavy metals. Chemically, dimercaprol binds to the arsenic in lewisite.