Paradoxically, cortisol promotes not only gluconeogenesis in the liver, but also glycogenesis. Cortisol also increases glycogen synthesis (glycogenesis) in the liver, storing glucose in easily accessible form. Refer to glycolysis for further information of the regulation of glycogenesis. The monosaccharide glucose-6-phosphate (G-6-P) is typically the input substance for glycogenesis. It regulates the storage of glycogen which it can form from glucose (glycogenesis). Therefore, PKB acts to start the process of glycogenesis, which ultimately reduces blood-glucose concentration. UDP is an important factor in glycogenesis. Glycogenesis refers to the process of synthesizing glycogen. Glycogenesis is the metabolic pathway in which glycogen is created. Glycogenesis occurs primarily in the liver, skeletal muscles, and kidney.