Immature neurons are more prone to synaptic depression than potentiation after high frequency stimulation. It modulates synaptic plasticity by enhancing the induction and maintenance of long-term potentiation. Blockade of the NMDA receptors prevents induction of long-term potentiation and impairs spatial learning. This process of synaptic strengthening is known as long-term potentiation. The morphogenesis of dendritic spines is critical to the induction of long-term potentiation (LTP). The synapse circuit demonstrated long-term potentiation for learning as well as inactivity based forgetting. This physiological effect is commonly referred to as post-activation potentiation, or the PAP effect. Such a physiological effect is commonly referred to as post-activation potentiation, or the PAP effect. In order to minimize this potentiation, postprandial administration (taken after meals) of moclobemide is recommended. PI3Ks have also been implicated in long-term potentiation (LTP). Loss of PrP function has been linked to long-term potentiation (LTP). This phenomenon was soon referred to as long-term potentiation (LTP). Hz helps strengthening the connection known as long-term potentiation. For more information, see long-term potentiation (LTP). They are one of the major factors in long-term potentiation.