Both SLC16A1 and SLC16A3 expression were increased in LGG and GBM.
Both SLC16A1 and SLC16A3 expression were increased in LGG and GBM. GO analysis indicated that SLC16A1 was related to Herpes simplex virus 1 infection while SLC16A3 was involved in neutrophil activation. SLC16A3 as well as SLC16A1 were closely related to immune infiltration and checkpoints LGG and GBM.